The Science & Practice of Perfecting Your Sleep | Huberman Lab Essentials
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Sleep is described as a complex physiological process essential for resetting brain and body health, broadly categorized into non-rapid eye movement (non-REM) sleep and rapid eye movement (REM) sleep. During REM sleep, voluntary muscles are paralyzed to prevent physical enactment of dreams, with the exception of the extraocular and inner ear muscles required for dreaming movements. The architecture of a typical night follows a cyclical pattern where deep slow-wave non-REM sleep predominates in the first half of the night, while lighter non-REM stages and increasing amounts of REM sleep characterize the second half. This distribution is critical because depriving individuals of early-stage deep sleep can lead to autonomic dysfunction and metabolic issues like dysregulated blood sugar, whereas later-stage REM deprivation affects cognitive functions such as learning, memory, emotional regulation, and testosterone production. Consequently, both phases are non-negotiable for optimal health; lacking either results in distinct profiles of mental and physical dysfunction that cannot be fully compensated by the other phase alone. The quality of sleep is just as important as quantity, a fact often compromised by common substances like caffeine and alcohol. Caffeine has an average half-life of five to six hours, meaning consuming it late in the day can block deep sleep for up to 30% and create a "tsunami" effect when adenosine builds up again after the stimulant wears off. Experts recommend cutting off caffeine intake eight to ten hours before bedtime to avoid this crash cycle. Similarly, while alcohol acts as a sedative that helps people fall asleep faster by lowering consciousness levels, it fragments sleep architecture and significantly blocks REM sleep. This leads to punctured awakenings throughout the night where individuals do not feel restored despite spending time in bed. Furthermore, substances like THC can also block REM sleep; when usage stops, the brain attempts to repay its "REM debt," often resulting in intense, bizarre dreams as it tries to recover lost restorative cycles that may never be fully regained. Melatonin supplementation is frequently misunderstood regarding its efficacy and dosage. While melatonin serves as a chemical signal from the pineal gland indicating day versus night via the suprachiasmatic nucleus, it does not generate sleep itself but rather initiates the process like an official starting a race. In healthy adults under age 60, studies show that supplementation only marginally increases total sleep time by about four minutes and efficiency by roughly two percent, making high-dose supplements (often 5 to 10 mg) largely unnecessary compared to natural production levels which are far lower. Supplementation is primarily beneficial for older adults whose pineal glands may calcify or function less efficiently over time. Rather than relying on exogenous molecules, the first line of defense should be behavioral tools such as viewing bright sunlight early in the day and avoiding screens late at night, alongside cognitive behavioral therapy for insomnia (CBT-I), which offers long-term benefits comparable to sleeping pills without dependency risks. Practical strategies for optimizing sleep include managing naps carefully and adopting unconventional habits like maintaining a consistent wake-up time even after poor nights of rest. Napping can offer cardiovascular and emotional regulation benefits if limited to 20 to 30 minutes, but individuals with insomnia should avoid them entirely as daytime sleepiness reduces the drive needed for nighttime sleep pressure. If one must nap, it is best done early in the day rather than late afternoon to prevent grogginess or interference with night-time rest. Additionally, establishing a wind-down routine similar to landing an airplane gradually helps transition from wakefulness to sleep, while writing down worries before bed can reduce time-to-sleep by 50%. Finally, removing clocks and phones from the bedroom prevents anxiety about waking up in the middle of the night; if one wakes up during fragmented sleep, checking the time only exacerbates distress without aiding return to rest. Ultimately, society should view getting adequate sleep as a civil right rather than a sign of slothfulness, empowering individuals to prioritize their biological needs through informed choices and behavioral adjustments.
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Welcome to Huberman Lab Essentials,
where we revisit past episodes for the
most potent and actionable science-based
tools for mental health, physical
health, and performance.
And now, my discussion with Dr. Matt
Walker. Let's start off very basic. What
is sleep?
Sleep is probably the single most
effective thing you can do to reset your
brain and body health. Sleep is a
process though is an incredibly complex
physiological ballet. Sleep is broadly
separated into these two main types and
we've got non-rapid eye movement sleep
on the one hand and then we've got rapid
eye movement sleep on the other. When
you go into REM sleep, you are
completely paralyzed. You are locked
into a physical incarceration of your
own body. Amazing. The brain paralyzes
the body so that the mind can dream
safely.
Because think about how quickly we would
have all been popped out of the gene
pool, you know, if I think I'm, you
know, one of the best skydivers who can
just simply fly and I get up on my
apartment window and I leap out. You're
done.
You're done. Now, of course, the
involuntary muscles, thankfully, aren't
um paralyzed so you keep breathing, your
heart keeps beating. You go through
these bizarre, what we call, autonomic
storms. There are only two voluntary
muscle groups that are spurred from the
paralysis. Bizarre. One, your
extraocular muscles because if they were
paralyzed, you wouldn't be able to have
rapid eye movements.
Mhm. And the other that we later
discovered was the inner ear muscle.
Some people have argued that the reason
the eyeballs are spurred from the
paralysis is because if your eyeballs
are left for long periods of time
inactive, you may get things such as
oxygen sort of issues in the um
aqueous or vitreous humor. And so, the
eyeballs have to keep the brain in some
way The systems of anterior eye are are
made to require movement.
People with glaucoma have deficits in in
drainage through the anterior chamber.
So, maybe take me through the arc of a
night. When I first fall asleep, I'll go
into the light stages of non-REM sleep,
stages one and two of non-REM, and then
I'll start to descend down into the
deeper stages of non-REM sleep. So,
after about maybe 20 minutes, I'm
starting to head down into stage three
non-REM and then into stage four non-REM
sleep.
And as I'm starting to fall asleep, as
I've cast off from the usually with me
murky waters of wakefulness,
and I'm in the shadows of sleep, stages
one and two, my heart rate starts to
drop a little bit. And then my brain
wave pattern activity starts to slow
down. Normally, when I'm awake, it's
going up and down maybe 20, 30, 40, 50
times a second.
As I'm going into light non-REM sleep,
it will slow down to maybe 15, 20, and
then really starts to slow down down to
about sort of 10 or eight cycles per
second, eight cycle waves per second.
Then as I'm starting to move into stages
three and four non-REM sleep, several
remarkable things happen. All of a
sudden, my heart rate really does start
to drop. Hundreds of thousands of cells
in my cortex all decide to fire together
and then they all go silent together.
And it's this remarkable physiological
coordination of the likes that we just
don't see at during any other brain
state. I will then stay there for about
another 20 or 30 minutes. So, now I'm
maybe 60 or 70 minutes into my first
sleep cycle.
And then I'll start to rise back up back
up into stage two non-REM sleep. And
then after about 80 or so minutes, I'll
pop up and I'll have a short REM sleep
period.
And then back down I go again, down into
non-REM, up into REM.
And you do that reliably, repeatedly,
and I will be doing that every 90
minutes. At least that's the average for
most adults. In the first half of the
night, the majority of those 90-minute
cycles are comprised of lots of deep
non-REM sleep. That's when I get my
stage three and four of deep non-REM
sleep.
Once I push through to the second half
of the night, now that seesaw balance
changes, and instead the majority of
those 90-minute cycles are comprised
either of this lighter form of non-REM
sleep, stage two non-REM sleep, and much
more and increasingly more rapid eye
movement sleep.
suffers more, those that lack the early
phase or those that lack the later phase
of the night? Depends on what you the
outcome measure is. For example, during
deep non-REM sleep, that's where we get
this It's almost a form of natural blood
pressure medication. And so when I take
that away from you, the next day,
usually going to see autonomic
dysfunction, we're usually going to see
abnormalities in heart rate, blood
pressure. We also know that it's during
deep non-REM sleep that there is a
certain control of specific hormones.
For example, we know that the insulin
regulation of sort of metabolism,
meaning how will you look from a
regulated blood sugar perspective versus
dysregulated, pre-diabetic look of
profile.
That's where deep sleep seems to matter.
If we selectively deprive you of that,
we can see um
Growth hormone is different actually. So
that's a that's a beautiful
demonstration where growth hormone seems
to be more REM sleep dependent, and
that's why we can come on to the effects
of alcohol. And there's there's some
really impressive, frightening data on
on alcohol and its disruption of um of
sleep. But then we also know
testosterone. You know, peak levels of
testosterone happen during REM sleep.
So, the second half of the night. It
Which is the second half of the night.
So, it really just means that the
your profile of mental and physical
dysfunction will be different under both
of those conditions. Which one would you
prefer? I would prefer neither of them,
and it really depends on what you're
trying to optimize for. So, it's it's
just so complicated. You know, sleep is
just so profoundly detrimental to us if
you were to take it at face value. You
know, you're not finding a mate, you're
not reproducing, you're not foraging for
food, you're not caring for your young,
and worst of all, you're vulnerable to
predation. On any one of those grounds,
sleep probably should have been selected
against. But, it wasn't. Sleep has
fought its way through heroically at
every step along the evolutionary path.
And therefore, every sleep stage has
also survived as best we can tell.
What that means is that those are
non-negotiable. If Mother Nature had
found a way to even just sort of, you
know,
thin slice some of that sleep from us,
there would have been fast, I'm sure,
evolutionary benefits, but it looks as
though she hasn't, and I'm usually in
favor of her wisdom after 3.6 million
years. So, in this arc of the night, uh
slow wave sleep predominates uh early in
the night, and then REM sleep.
There's a scenario that many people,
including myself, experience on a
regular basis, which is
they go to sleep, sleeping just fine, 3
4 hours into it, they wake up.
They wake up for whatever reason. Maybe
there was a noise, maybe the temperature
isn't right. We will certainly talk
about sleep hygiene, etc.
They get up, they go to the restroom.
They might flip on the lights, they
might not. They go back to sleep. Let's
say after about 10 15 minutes, they're
able to fall back asleep.
How detrimental is that wake up
episode or or um
event in terms of
longevity, learning, etc. It is
perfectly natural and normal,
particularly as we progress with age. At
the end of our REM sleep period of the
90-minute cycle, almost everybody wakes
up and we make a postural movement. We
turn over because we've been paralyzed
for so long and the body will also like
to shift. For the most part, I think we
can be more relaxed about that. Where we
have to be a bit more attentive though
is if you are spending long periods of
time not being able to get back to sleep
and usually we define that by saying if
it's been 20 25 minutes. The other thing
is if it's happening very frequently.
So, even if you're um
you know, not awake for 25 minutes
stretches, but you're finding yourself
waking up and being consciously aware
that you've woken up for maybe six,
seven, or eight times throughout the
night and your sleep is very what we
call fragmented.
The great science of sleep in the past 5
or 10 years has has been yes, quantity
is important, but quality is just as
important and you can't have one without
the other in terms of a good beneficial
next day outcome.
You can't just get 4 hours of sleep but
brilliant quality of sleep and be
unimpaired. Nor can you get 8 hours of
sleep but have very poor quality of
sleep and be unimpaired the next day.
I'm a big proponent of people getting
uh some sunlight, ideally sunlight, but
other forms of bright light into their
eyes early in the day and when they want
to be awake. Yep.
Essentially during the phase of their
24-hour circadian cycle when temperature
is rising and then starting to less
light in their eyes
as our temperature's going down in term
of later in the day and in the evening.
I think that's exactly what we recommend
right now, which is try to get at least
30 to 40 minutes of exposure to some
kind of natural daylight. There was some
great work recently coming out in the
occupational health domain where they
moved workers from offices that were
just facing walls and you know, didn't
have any exposure to natural daylight
and then they did a time period during
that study where they actually were in
front of a window and working and they
measured their sleep. And their sleep
time and their sleep efficiency
increased quite dramatically. I think
the increase in total sleep time was
well over 30 minutes and the improvement
in sleep efficiency was 5 to 10%. You
know, and if you're batting an 80%, you
know, sleep efficiency average, we're a
bit concerned about that. But add 10% to
that and now you're in, you know, a
great echelon of healthy sleepers. These
portals are the only way to to
convey to the rest of the brain and body
about the time of day and wakefulness. I
have a number of questions about
caffeine. Does the timing in which we
ingest caffeine
play an important role in whether or not
it works for us or against us? The dose
and the timing makes the poison.
Caffeine has a half-life and it's
metabolized. The half-life is somewhere
between 5 to 6 hours and the
quarter-life therefore somewhere between
10 to 12 hours. It's variable. Different
people have different durations of its
action, but for the average adult 5 to 6
hours. So, let's say that, you know,
I've been awake for 12 hours now and
it's, you know, 8:00 p.m. and I'm
feeling a bit tired, but I want to push
through and I want to keep working for
another couple of hours. So, I have a
cup of coffee. All of a sudden, I was
feeling tired, but I don't feel like
I've been awake for 12 hours anymore.
Then, after a few hours and the caffeine
is starting to come out of my system,
not only am I hit with the same levels
of adenosine that I had before I'd had
the cup of coffee several hours ago,
it's that plus all of the adenosine
that's been building up during the time
that the caffeine has been in my system.
It's sort of a an avalanche of It is a
tsunami wave.
Yeah. And I have a caffeine crash. Given
somebody who typically gets into bed
around 10 10:30 and falls asleep around
uh 11 11:30, Yeah.
when would you recommend they halt
caffeine intake? And I These are not um
strict prescriptions, but I think people
do benefit from having some uh fairly
clear guidelines of what might be might
work for them. Would you say cut off
caffeine by
what time of the day? I would usually
say take your typical bedtime and count
back 10 hours or 8 hours of time. That's
the time when you should really stop,
you know, using caffeine is the
suggestion. And the reason is because
for those people who even just keep
drinking up until, you know, the into
the evening,
you're right that they can fall asleep
fine, maybe they stay asleep, but the
depth of their deep sleep is not as deep
anymore.
And so, there are two consequences. The
first is that for me and it can be up to
by 30% and for me to drop your deep
sleep by 30%, I'd have to age you by
between 10 to 12 years
or you can just do it every night to
yourself with a couple of espressos.
The second is that you then wake up the
next morning and you think, "Well, I
didn't have problems falling asleep and
I didn't have problems staying asleep,
but I don't feel particularly restored
by my sleep." So, now I'm reaching for
three or four cups of coffee the next
morning rather than just two or three
cups of coffee. And so goes this
dependency cycle that you then need your
uppers to wake you up in the morning and
then sometimes people will use alcohol
in the evening to bring them down
because they're overly caffeinated and
alcohol, and we can speak about that
too, also has very deleterious impacts
on your sleep as well. Caffeine and
alcohol represent the uh they're kind of
two opposite ends of the spectrum. What
happens when somebody has a glass We
always hear a glass or two of wine in
the evening or a
a cocktail after dinner? How does that
impact their sleep? So, alcohol, if
we're thinking about classes of drugs,
they're in a class of drugs that we call
the sedatives.
It's sedating your cortex.
And sedation is not sleep. But when we
have a couple of drinks in the evening,
when we have a couple of nightcaps, we
mistake sedation for sleep saying,
"Well, I always when I have a like a
couple of whiskeys or a couple of
cocktails, it always helps me fall
asleep faster."
In truth, what's happening is that
you're losing consciousness quicker,
but you're not necessarily falling
naturalistically asleep any quicker. So,
that's one of the first sort of things
just to keep in mind. The second thing
with alcohol is that it fragments your
sleep. And we spoke about the quality of
your sleep being just as important as
the quantity.
And alcohol through a variety of
mechanisms, some of which are activation
of that autonomic nervous system, that
fight or flight branch of the nervous
system,
alcohol will actually have you waking up
many more times throughout the night.
So, your sleep is far less continuous.
Now, some of those awakenings will be of
conscious recollection the next day.
You'll just remember waking up. Many of
them won't be.
And so, but yet your sleep will be
littered with these sort of punctured
awakenings throughout the night. And
again, when you wake up the next
morning, you don't feel restored by your
sleep. The third part of alcohol in
terms of an equation is that it's quite
potent at blocking your REM sleep, your
rapid eye movement sleep. And REM sleep
is critical for a variety of cognitive
functions, um some aspects of learning
and memory. It seems to be critical for
aspects of emotional and mental health.
It's overnight therapy. What we've
discovered over the past 20 years here
at the sleep center is that
there is no major psychiatric disorder
that we can find
in which sleep is normal.
And so I think that firstly told us
there is a very intimate association
between your emotional mental health and
your sleep health. I don't want to be
puritanical here, you know, I'm just a
scientist and I'm not here to tell
anyone how to live. All I'm trying to do
is empower people with some of the
scientific literature regarding sleep
and then you can make whatever informed
choices that you want. My job is not to
to tell people um that prescription for
life. It's just to offer some scientific
information. I would like to ask about
marijuana. In many places, not all,
medical marijuana is approved or is
legal. Does marijuana disrupt sleep? THC
can
s- seems to speed up the time with which
you fall asleep. But again, if you look
at the the electrical brain wave
signature of your falling asleep with
and without that THC, it's not going to
be an ideal fit. So you could argue it's
non-natural. It too, but through
different mechanisms, seems to block REM
sleep. And that's why a lot of people
when they're using will tell me, "Look,
you know, I I definitely I was dreaming
I don't remember, you know, many of my
dreams." And then when they stop using
uh THC, they'll say, "I was having, you
know, just crazy, crazy dreams." And the
reason is because there is a rebound
mechanism. REM sleep is very clever and
alcohol is the same way in this sense.
It's the same homeostatic mechanism.
Some people will tell me, "Look, if I
have a bit of a wild Friday night with
some alcohol, you know, maybe I'll sleep
late into the next morning, and I'll
just have these really intense dreams.
So, and I thought I wasn't having any
REM sleep." Well, the way it works is
that it's during in the middle of the
night, really, um when alcohol blocks
your REM sleep.
And your brain is smart. It understands
how much REM sleep you should have had,
how much REM sleep you have not because
the alcohol's been in the system, and
finally, in those early morning hours,
when you're getting through to sort of,
you know, 6, 7, 8 a.m., all of the
sudden, your brain not only goes back to
having the same amount of REM it would
have had, it does that, plus it tries to
get back all of the REM sleep that it's
lost. Does it get back all of the REM
sleep? No, it doesn't. It never gets
back all of the REM sleep, but it tries.
And so, you have these really intense
periods of REM sleep, hence, you have
really intense, bizarre dreams. And
that's what happens also with THC. You
build up this pressure for REM sleep,
this debt for REM sleep. Will you ever
pay it back?
It doesn't seem as though you get back
everything that you lost, but will you
get back some of it? Yes, the brain will
start to devour more because it's been
starved of REM sleep for so long. So,
I'd love to chat for a moment about the
kind of grand the original, I should
say, that not the granddaddy, but the OG
of sleep supplementation, which is
melatonin.
Yeah. I was always taught, and I'm
assuming it's still true, that the only
source of melatonin in the brain and
body is the pineal gland. Is that still
true? It Yeah, it seems to be from best
that we can tell.
I have to imagine we have melatonin
receptors in the brain and body.
It's correct. Essentially, your brain
has a central master 24-hour clock
called the suprachiasmatic nucleus that
keeps internal time. Now, it knows
24-hour time,
but it needs to tell the rest of the
brain and the body the 24-hour time as
well. And one of the ways that it does
this is by communicating a chemical
signal of 24-hour nurse
of light and day using this hormone
melatonin. And when it is at low levels
or it's non-existent, it's communicating
the message it's daytime. And for us,
diurnal species, it says it's time to be
awake.
Yet, at nighttime when dusk approaches
and the brake comes off melatonin and we
start to release it, then it signals to
the rest of the brain and the body,
"Look, it's dusk and it's nighttime and
for us, diurnal species, it's time to
think about sleep." So, melatonin
essentially tells the brain and the body
when it's day and when it's night and
with that, when it's time to sleep, when
it's time to wake. But it doesn't really
help with the generation of sleep itself
and this is why we'll come on to what
those studies of supplementation have
taught us. So, it tells the rest of my
brain and body it's time to go to sleep.
It perhaps even aids with the transition
to sleep, but it's not going to, for
instance, ensure the overall structure
of sleep or
it's not the conductor that's guiding
the sleep orchestra, so to speak,
throughout the entire night. You know,
melatonin is like the starting official
at the 100-meter race in the Olympics.
That's a better analogy. Call calls all
of the the sleep races to the line and
it begins the great sleep race, but it
doesn't participate in the race itself.
That's a whole different set of brain
chemicals and brain and brain regions.
Which then brings us on to perhaps the
question of supplementation,
which is
is it helpful for my sleep? Will I sleep
longer? Will I sleep better? Sadly, the
evidence in healthy adults who are not
older age suggests that melatonin is not
really particularly helpful as a sleep
aid. I think there's a recent
meta-analysis. And what that
meta-analysis told us is that melatonin
will only increase total amount of sleep
by 3.9 minutes on average. Minutes?
Minutes.
And it will only increase your sleep
efficiency by 2.2%
So, it the the source is not strong. The
force is not strong in in in this one
when it comes to
a tool that in healthy people
who are not of older age,
it doesn't seem to be especially
beneficial. Now, you know, results can
vary. Everyone is different, of course.
So, we're talking about the average, the
so-called average human adult here.
Well, melatonin, um, in defense of what
you're saying, and also I should mention
I have a colleague at Stanford, uh,
Jamie Zeitzer, who I know
Chuck Zeitzer's lab at Harvard where he
also trained, a terrific sleep
researcher, and I asked him about
melatonin, and he essentially said the
same thing that you just said, which is
very little, if any, evidence that it
can improve sleep, and yet it's probably
the most, um, commonly consumed
so-called sleep aid.
Hundreds of millions of dollars
industry. The only population where we
typically see some benefit, and it often
is prescribed, is in older adults.
Because as we get older you can
typically have what's
called calcification of the pineal
gland, which means that that gland
that's releasing melatonin doesn't work
as well anymore. That's why older adults
can have problems falling asleep or
staying asleep. It's not the only
reason, by sure any stretch of the
imagination, but it's one of the
reasons, and it's why melatonin
supplementation in those cohorts, older
adults, especially older adults with
insomnia, people have thought about that
as maybe an appropriate use case. Do we
know how much melatonin is released into
the bloodstream per night? Um, And can
we use that as a kind of a rule of thumb
by which to compare the typical amount
that someone would supplement? I mean,
typically the supplements for melatonin
that I see
in the pharmacy and elsewhere and online
range anywhere from 1 mg to 12 or even
20 mg.
Yep.
My guess is that a normal night's
release of melatonin typical for
somebody in their 20s, 30s, 40s would be
far lower than that. Am I correct or
wrong? Yeah, it's it's a many magnitudes
lower and this is one of the problems is
that I see that, too. I see, you know,
typical doses are, you know, 5 mg or 10
mg and of course, you know, if you're a
supplement company, you know, putting 10
mg versus 5 mg, if that's what you're
actually doing, you know, it's kind of
like the super gulp size. You know,
nobody wants to lower price. They just
want you to, you know, we'll just give
you more for the same price and that's
how we'll compete. So, it's been this
escalating arms race of melatonin
concentration. And it really does not
look meaningful for
you know, for sleeping in any way. What
we've actually found is that the optimal
doses for where you do get sleep
benefits in the populations that we've
looked at are somewhere between 0.1 and
0.3
mg of melatonin. In other words, the
typical doses are usually 10 times, 20
times, maybe more than what your body
would naturally expect. And this is what
we call a supra physiological dose. In
other words, it's far above what is
physiologically normal. I like to think
in terms of of manipulating any aspect
of our biology that behavioral tools
always are the first line of entry.
Yeah. Then nutrition. Everyone has to
eat sooner or later, if you're fasting.
Then perhaps supplementation, then
prescription drugs, and then perhaps
brain machine interface devices that you
use to induce something. And those could
be done in combination, but what
concerns me is when I hear people uh
say, "Well, what should I take?" without
thinking about their behavior, their
light viewing behavior, et cetera. But,
of course, these things work in
combination. When it comes to sleep,
there are many low-hanging fruits that
don't necessarily require you to, you
know, put
sort of exogenous molecules, in other
words, things like supplements into your
body or, you know, use different types
of drugs to help you get there. Now,
when it comes to prescription sleep
aids,
I think I've been, again, a little bit
too forthright.
We know in clinical practice that there
may be a time and a place for things
like sleeping pills. They are a
short-term solution to certain forms of
insomnia, but they are not recommended
for the long term. And we also know that
there are lots of other ways that you
can get a sleep um
help, or you can get a sleep curative
profile from things like cognitive
behavioral therapy for insomnia, which
is a non-drug approach, psychological
effective from what I understand.
Very just as effective as sleeping
pills, great data, more effective in the
long term. There's a recent study
published that after working with that
therapist, some of the benefits lasted
almost a decade, you know. Now, if you
stop sleeping pills, usually you have
rebound insomnia, where your sleep goes
back to being just as bad if not worse.
And I think the same is true when we
think about supplementation. There are
so many things that are easy to
implement when it comes to sleep that
don't require venturing out into those
waters. And again,
we're not here to tell anyone about
whether they should venture or not.
That's completely your choice. All I'm
saying is that if you want to think
about optimizing your sleep, there are a
number of ways that you can do it that
don't necessarily require you to swallow
anything cool or inject anything cool or
you know, smoke anything cool. Right.
And for which the margins of safety are
quite quite wide. Let's talk about naps.
I love naps. I come from a long history
of nappers. What are the data on naps?
And what are your thoughts about keeping
naps short, meaning 20 to 30 minutes
versus getting out past 90 minutes, 2
hours? Yay, nay, or meh? Naps can have
some really great benefits. We found
benefits for cardiovascular health,
blood pressure for example. We found
benefits for levels of cortisol. We
found benefits for learning and memory
and also emotional regulation. How long
are the naps typically in those studies?
Anywhere between 20 minutes to 90
minutes. Sometimes we like to use a
90-minute window so that the participant
can have a full cycle of sleep and
therefore they get both non-REM and REM
sleep within that time period. And then
we correlate how much benefit did you
get from the nap and how much of that
benefit was explained by what REM sleep
you got, what deep sleep you got, what
light sleep you got. What we've also
found is that naps of as little as 17
minutes can have some quite potent
effects on for example learning.
None of this is novel. NASA pioneered
this back in the 1990s.
And during the missions they were
experimenting with naps for their
astronauts. And what they found was that
naps of little as 26 minutes improved um
uh mission performance by 34%
and improved daytime alertness by 50%.
And it birthed what was then called the
NASA nap culture throughout all
terrestrial NASA staff during that time
period. So it's long been known that
naps can have a benefit. Naps however
can have a double-edged sword. There is
a dark side to naps. When you nap, you
are essentially opening the valve on the
pressure cooker of sleep pressure,
and some of that sleepiness is lost by
way of the nap. Some people, however, if
they are struggling with sleep at night
and they nap during the day, it makes
their sleep problems even worse. So, for
people with insomnia, we typically
advise against napping.
And the advice is if you can nap
regularly and you don't struggle with
sleep at night, then naps are just fine.
But if you do struggle with sleep, stay
away from naps. If you are going to nap,
try to limit your naps. Try to cut them
off a bit like sort of caffeine, maybe,
you know, 8 to sort of 12 hours, maybe
not that, you know, far off, maybe sort
of 7 to 6 hours is a good rule of thumb.
Try not to nap essentially late in the
afternoon. And if you do take a nap
and you want to maintain your You don't
want to have that grogginess hangover
that can happen after a full night of
sleep for the first hour, try to limit
it to about 20 25 minutes. And that way
you don't go down into the very deepest
stages of sleep, which I If I wrench you
out of with an alarm, you almost feel
worse. No one should feel guilty about
getting the sleep that they need. And I
think that's been one of the big
problems in society. Society has
stigmatized sleep with these labels of
being slothful or lazy. And we're almost
embarrassed, you know, to tell
colleagues that we we take a nap. I
think sleep is a right of human beings.
And I therefore think that sleep is a
civil right of all human beings. And no
one should make you feel unproud of
getting the sleep that you need. Are
there any unconventional sleep tips, the
what if kind of things that Yeah, what
if
it turns out that And I here I just I
I've got a blank there for you to fill
in. The first one which is
unconventional along the lines of naps,
if you've had a bad night of sleep, do
nothing.
What I mean by that is don't wake up any
later, don't sleep in the following day
to try and make up for it. Don't nap
during the day.
Don't consume extra caffeine to wake you
up to try to get you through the day and
don't go to bed any earlier to think
that you're going to compensate. If you
wake up later, you're not going to be
sleepy until later the evening. So,
you're going to go to bed at your normal
time and you won't be sleeping. You'll
think, "Well, I just came off a bad
night of sleep and now I I still I'm I
can't even get to sleep and it's my
normal time." It's because you slept in
later than you would otherwise and you
reduce the window of adenosine
accumulation before your normal bedtime.
So, don't go don't wake up any later.
Don't use more caffeine for the reasons
that that are obvious because that's
only going to crank you and keep you
awake the following night or decrease
the probability of a good following
night of recovery sleep. And then
finally, don't go to bed any earlier.
Resist and resist and go to bed at your
normal time. What I want to try and do
is prevent you from thinking, "Well, I
had such a bad night last night and I
normally go to bed at 10:30. I'm just
going to get into bed at 9:00. My body
is not ready to to sleep at 9:00, but
I'm worried because I had a bad night of
sleep last night. So, I get into bed and
now I'm tossing and turning for the
first hour and a half because it's not
my natural sleep window, but I just
thought it was a good idea." I think the
second tip um I would offer in terms of
unconventional is have a wind down
routine.
Many of us think of sleep as if it's
like a light switch that we just jump
into bed and when we turn the light out,
sleep should arrive in that same way.
Sleep is It's
process. It's much more like landing a
plane. It takes time to gradually
descend down onto the terra firma of
what we call good solid sleep at night.
Find out whatever works for you and it
could be light stretching. I usually
meditate for about 10 or 15 minutes
before bed. Some people like reading.
Try not to watch television in bed.
That's usually advised against.
to your
Too much light too activating. You know,
you wouldn't race into your garage and
come to a a
screeching halt from 60 miles an hour.
You typically downshift your gears and
you slow down as you come into the
garage. It's the same thing with with
sleep, too. The other thing about
sort of that idea of shifting focus away
from your mind itself. Get your mind off
itself is a good piece of advice.
Catharsis. You can try to write down all
of the concerns that you have.
And do this not right before
but usually an hour or two before bed.
Some people call call it a worry
journal. And to me it's a little bit
like closing down all of the emotional
tabs on my browser. Because if I shut
the computer down and all of those tabs
are still open, I'm going to come back
in the morning and the computer's red
hot, the fan's going because it didn't
go to sleep because it couldn't because
there were too many tabs active and
open. I at first thought this just
sounds like hooey. It sounds very
Berkeley. It's kind of come by our we
all hold hands and you know, walk home
at the end of the day. But then this the
data started coming out. Really good
studies from good people and they found
that keeping one of those journals
decreased the time it takes you to fall
asleep by 50% 5-0. You know, it's well
on par with any pharmaceutical agent. I
think the fourth sort of little tip I
would give that's unconventional is
remove all clock faces from your bedroom
including your phone. Including your
phone because if you are having, you
know, a tough night,
knowing that it's 3:22 in the morning or
it's 4:48 in the morning does not help
you in the slightest. And it's only
going to make matters worse than better.
Matt, this has been an amazing deep dive
on sleep. It is chock-a-block full of
valuable takeaways. It's been a
a tremendously fun for me to dissect out
this incredible aspect of our lives that
that we call sleep with a fellow
scientist and a
fellow public educator. And
we don't just want to hear more from
Matt Walker. I speak for many people.
We need to. The work you're doing is
both
influential, but more importantly, it is
important work. It it has the impact
that's needed especially in this day and
age where science and medicine, public
health, and the the issues of the world,
etc., are really converging. So,
I know I speak on behalf of a tremendous
number of people when I just say, "Thank
you for doing the work you do and for
being you."